antagonist muscle वाक्य
उदाहरण वाक्य
मोबाइल
- Old adults often show heightened antagonist muscle coactivation during goal directed movement.
- If a motion is reversed, agonist and antagonist muscles switch roles.
- This excitatory interneuron innervates the alpha motor neuron that controls the antagonist muscle.
- When the agonist muscle is inhibited from contracting, the antagonist muscle contracts.
- There are co-contractions of antagonist muscles.
- Electrical stimulation to the antagonist muscles or vibrations has also been used with some success.
- A small device that can help in developing the antagonist muscles to those used while gripping with the hand.
- In others, a botulinum toxin injection into an extraocular muscle is accompanied by a bupivacaine injection into the antagonist muscle.
- The interposed nucleus is responsible for coordinating agonist / antagonist muscle pairs, and therefore a lesion in this area causes tremor.
- If bupivacaine injection is combined with a small dose of botulinum toxin in the antagonist muscle, eye deviation during regeneration is minimized.
- The sensory input from the quadriceps also activates local interneurons that release the inhibitory neurotransmitter glycine onto motor neurons of antagonist muscles, blocking the innervation of these antagonistic ( hamstring ) muscles.
- In Wedensky s laboratory, and at the same time as Charles S . Sherrington ( 1857-1952 ), Beritashvili used the string galvanometer to study the central coordination of spinal reflexes in the registration of action currents of antagonist muscles.
- Injection of small dose of botulinum toxin in the antagonist muscle weakens it for a few weeks, preventing stretching of the bupivacaine-injected muscle, allowing it to regenerate shorter than otherwise, thereby providing about twice the alignment correction of bupivacaine alone.
- The cross-connectivity between areas that are normally segregated in the sensory cortex may prevent normal sensorimotor feedback and so contribute to the observed co-contraction of antagonist muscle groups, and inappropriately timed and sequenced movements that underlie the symptoms of focal dystonia.
- Although cardiac muscle and smooth muscle are not directly connected to the skeleton, they also have tonus in the sense that although their contractions are not matched with those of antagonist muscles, the non-contractile state is characterized by ( sometimes random ) enervation.
- Second is the operation of Sherrington's Law describing reciprocal inhibition between agonist-antagonist muscle pairs, and by implication the stretching of extraocular muscle that must occur whenever Sherrington's Law is made to fail, thereby causing an unrelaxed ( contracted ) muscle to be stretched.
- When confronted to an unexpected slip or trip during walking, compared to young adults, old adults have a less effective balance strategy : smaller and slower postural muscle responses, altered temporal and spatial organization of the postural response, agonist-antagonist muscles coactivation and greater upper trunk instability.
- Neurological signs and symptoms include cerebellar ataxia ( unsteady walking with uncoordinated limb movements ), dysarthria ( slurred speech ), dysphagia ( difficulty in swallowing ), tremor, epilepsy ( both partial and generalized ), vertical supranuclear palsy ( upgaze palsy, downgaze palsy, saccadic palsy or paralysis ), sleep inversion, gelastic cataplexy ( sudden loss of muscle tone or drop attacks ), dystonia ( abnormal movements or postures caused by contraction of agonist and antagonist muscles across joints ), most commonly begins with in turning of one foot when walking ( action dystonia ) and may spread to become generalized, spasticity ( velocity dependent increase in muscle tone ), hypotonia, depression that can include hallucinations, delusions, mutism, or stupor.
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